Clinical Management of Circulatory Shock Quiz

Distributive shock is often triggered by widespread vasodilation, as seen in sepsis.

Inadequate perfusion in cardiogenic shock affects vital organs, with the brain being a primary target.

Epinephrine is a potent vasopressor that helps increase blood pressure and improve perfusion in shock.

Hypovolemic shock often results in decreased urine output as the body conserves fluids.

Beta-blockers, with their negative inotropic effects, are contraindicated in shock management.

Impaired cardiac function in cardiogenic shock leads to congestion in the pulmonary circulation.

Hypovolemic shock requires immediate fluid resuscitation to restore intravascular volume.

Unlike other forms of shock, septic shock typically presents with tachycardia rather than bradycardia.

Neurogenic shock, often associated with spinal cord injuries, manifests with bradycardia due to loss of sympathetic tone.

Obstructive shock results from mechanical obstruction of blood flow, commonly seen in conditions like pulmonary embolism.

Fluid resuscitation aims to enhance cardiac output by restoring intravascular volume.

Cardiogenic shock often presents with elevated central venous pressure due to impaired cardiac function.

Dobutamine enhances the force of myocardial contractions, addressing the decreased cardiac output in cardiogenic shock.

Distributive shock often leads to impaired tissue oxygenation, reflected in elevated lactate levels.

Norepinephrine acts as a potent vasoconstrictor, enhancing systemic vascular resistance in shock.

Epinephrine acts as a potent vasopressor, increasing systemic vascular resistance to improve perfusion in shock.