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ACLS Bradycardia Management Quiz

#1

During transcutaneous pacing in ACLS, what is the recommended initial pacing rate?

100 beats per minute
Explanation

The recommended initial pacing rate during transcutaneous pacing in ACLS is 100 beats per minute to address symptomatic bradycardia effectively.

#2

In ACLS, what is the primary goal of treating bradycardia?

To increase heart rate
Explanation

The primary goal of treating bradycardia in ACLS is to increase heart rate and improve cardiac output, enhancing perfusion to vital organs.

#3

What is the primary mechanism of action of atropine in managing bradycardia?

Negative chronotropy
Explanation

The primary mechanism of action of atropine in managing bradycardia is negative chronotropy, inhibiting vagal influences and increasing heart rate.

#4

In bradycardia, what is the primary effect of atropine on the heart?

Increases heart rate
Explanation

In bradycardia, the primary effect of atropine on the heart is to increase heart rate by blocking vagal influences and promoting sympathetic activity.

#5

In ACLS, what is the primary mechanism of action of dopamine in managing bradycardia?

Positive inotropy
Explanation

The primary mechanism of action of dopamine in managing bradycardia in ACLS is positive inotropy, enhancing myocardial contractility and increasing heart rate.

#6

Which drug is the first-line treatment for symptomatic bradycardia in ACLS?

Atropine
Explanation

Atropine is the first-line drug for treating symptomatic bradycardia in ACLS, acting by blocking vagal effects and increasing heart rate.

#7

In ACLS bradycardia management, at what rate should atropine be administered?

0.5 mg every 3-5 minutes
Explanation

Atropine should be administered at a rate of 0.5 mg every 3-5 minutes in ACLS bradycardia management to achieve the desired effect.

#8

Which medication is indicated for bradycardia caused by beta-blocker or calcium channel blocker toxicity in ACLS?

Glucagon
Explanation

Glucagon is indicated for bradycardia caused by beta-blocker or calcium channel blocker toxicity in ACLS, offering an alternative mechanism to increase heart rate.

#9

What is the recommended dose of dopamine in ACLS bradycardia management?

10-20 mcg/kg/min
Explanation

The recommended dose of dopamine in ACLS bradycardia management is 10-20 mcg/kg/min, providing positive inotropic support to increase heart rate.

#10

In ACLS bradycardia management, what is the recommended dose of atropine for a patient with a heart rate less than 60 beats per minute?

1 mg
Explanation

For a patient with a heart rate less than 60 beats per minute in ACLS bradycardia management, the recommended dose of atropine is 1 mg to prompt an increase in heart rate.

#11

Which of the following is a reversible cause of bradycardia in ACLS?

All of the above
Explanation

Various reversible causes such as hypovolemia, hypoxia, and acidosis contribute to bradycardia in ACLS, making 'All of the above' the correct answer.

#12

What is the recommended dose of epinephrine in symptomatic bradycardia according to ACLS guidelines?

0.5 mg every 3-5 minutes
Explanation

The recommended dose of epinephrine in symptomatic bradycardia according to ACLS guidelines is 0.5 mg every 3-5 minutes, aiming to increase heart rate.

#13

In ACLS, which method is considered the most effective for managing bradycardia with inadequate perfusion despite atropine?

Transcutaneous pacing
Explanation

Transcutaneous pacing is considered the most effective method in ACLS for managing bradycardia with inadequate perfusion despite atropine, directly controlling the heart rate.

#14

What is the recommended initial energy setting for transcutaneous pacing in ACLS?

100 mA
Explanation

The recommended initial energy setting for transcutaneous pacing in ACLS is 100 mA to establish effective pacing and address symptomatic bradycardia.

#15

Which of the following is NOT a reversible cause of bradycardia in ACLS?

Conduction system disease
Explanation

Conduction system disease is NOT a reversible cause of bradycardia in ACLS, as it pertains to intrinsic abnormalities in the heart's electrical conduction.

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